Medical Ozone Treats Lyme Disease and MSIDS



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Medical Ozone Therapy Information: Dr Shallenberger




Hope ozone generator



Chronic Lyme disease and MSIDS: These chronic infections demonstrate the inability of the patient’s immune system to defend itself against bacteria, fungi and viruses. 
These include Hepatitus B & C, Epstein-Barr virus, Cytomegalovirus, HIV, mycoplasma, tuberculosis, coxsackie virus, yeast, Borrelia (Lyme disease) and others.

These infections often respond to ozone therapy as ozone stimulates the immune system by as much as 400% to produce the cytokine molecules that it uses to fight infections.

The treatment protocol for any infection is rectal insufflation twice a day until the infection resolves and then three times a week for an additional 6 weeks to prevent a recurrence. Ozone can also be infused into the ears with Hope’s headphones.

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Ozone Therapy is a medical therapy that has been used worldwide for over 50 years with dramatic success and safety. In North America Ozone Therapy has not yet been fully recognized by the medical authorities, however if you review our ever growing list of Ozone Doctors, you will see that it is being adopted more and more by formally ‘traditional’ medical practitioners. It is clear that Ozone Therapy works, and it is becoming one of the most useful tools in medical clinics across America and around the World. (See “A Review of Ozone Therapy Applications” to learn how ozone is being administered).What does it do? To briefly summarize, ozone accomplishes these very important tasks in your body:

1/ Ozone is AntiAging (some of those AntiAging effects can be attributed to the following list of actions…)
2/ Ozone Increases Oxygenation of your Cells (it has been proven that cancer and disease grow in poorly oxygenated tissues in your body).
3/ Ozone Modulcates your Immune System (for those with a weakened immune system, Ozone will boost the immune system. For those with Auto-Immune Disorders, Ozone will modulate the immune system to help to stop it from attacking healthy human cells.)
4/ Ozone Increases Energy Production in your Cells (your cells need energy to be healthy; low energy levels mean that you and your cells will not be healthy and will age)
5/ Ozone Increases the Activity of your “Anti-Oxidant Enzyme Systems”. This means ozone will reduce the oxidation levels of your body.
6/ Ozone Reduces the level of acidity of your body (never mind the Alkaline Water…use Ozone!)
7/ Ozone kills Bacteria, Viruses (and virtually all other disease causing organisms) on contact
8/ Ozone Kills some Cancer cells on contact


Identification of novel activity against Borrelia burgdorferi persisters using an FDA approved drug library.


Although antibiotic treatment for Lyme disease is effective in the majority of cases, especially during the early phase of the disease, a minority of patients suffer from post-treatment Lyme disease syndrome (PTLDS). It is unclear what mechanisms drive this problem, and although slow or ineffective killing of Borrelia burgdorferi has been suggested as an explanation, there is a lack of evidence that viable organisms are present in PTLDS. Although not a clinical surrogate, insight may be gained by examining stationary-phase in vitro Borrelia burgdorferi persisters that survive treatment with the antibiotics doxycycline and amoxicillin. To identify drug candidates that can eliminate B. burgdorferi persisters more effectively, we screened an Food and Drug Administration (FDA)-approved drug library consisting of 1524 compounds against stationary-phase B. burgdorferi by using a newly developed high throughput SYBR Green I/propidium iodide (PI) assay. We identified 165 agents approved for use in other disease conditions that had more activity than doxycycline and amoxicillin against B. burgdorferi persisters. The top 27 drug candidates from the 165 hits were confirmed to have higher anti-persister activity than the current frontline antibiotics. Among the top 27 confirmed drug candidates from the 165 hits, daptomycin, clofazimine, carbomycin, sulfa drugs (e.g., sulfamethoxazole), and certain cephalosporins (e.g. cefoperazone) had the highest anti-persister activity. In addition, some drug candidates, such as daptomycin and clofazimine (which had the highest activity against non-growing persisters), had relatively poor activity or a high minimal inhibitory concentration (MIC) against growing B. burgdorferi. Our findings may have implications for the development of a more effective treatment for Lyme disease and for the relief of long-term symptoms that afflict some Lyme disease patients.


Borrelia burgdorferi; FDA approved drug library; SYBR Green I; drug discovery; persisters

Med Hypotheses. 1998 Jan;50(1):67-80.

Selective compartmental dominance: an explanation for a noninfectious, multifactorial etiology for acquired immune deficiency syndrome (AIDS), and a rationale for ozone therapy and other immune modulating therapies.


The most widely accepted etiological explanation for acquired immune deficiency syndrome (AIDS) currently invokes an infectious model involving the human immunodeficiency virus (HIV). Because this infectious model has failed to meet any conventional criteria for establishing microbial causation, this theory still relies on the high, though not perfect, statistical correlation linking presence of HIV antibodies with patients diagnosed with, and at risk for the syndrome. Many scientists and clinicians now doubt the HIV theory, though, and propose instead a multifactorial causation similar to that seen in cancer and heart disease. In order to discard the HIV model, however, it is necessary to explain the high statistical correlation mentioned above. Recent studies involving cellular mediated immunity and cytokine modulation may explain this statistical relationship without the need to invoke infectious causation, by suggesting certain functional characteristics and feedback loops in the immune system which the author calls selective compartmental dominance (SCD). SCD provides a model in which chronic dominance of the humoral immune compartment secondary to chronic high-dose antigenic challenge results in chronic suppression of the cellular immune compartment. This model predicts that even HIV-negative members of the risk groups are susceptible to AIDS, assigns no special causal role for HIV in AIDS, and suggests a rational course of nontoxic therapy that can potentially reverse cases in the earlier stages.

[Indexed for MEDLINE]