Lyme Disease Co-Infections

Is it normal to be losing your memory as you get older? Is it normal to have an Alzheimer’s
epidemic affecting not only the United States and also the rest of the world? Or is it  possible that there are multiple etiologies at the root of these conditions? We find that the majority of lyme patients with co- infections have severe memory and concentration problems.

Evidence of the connection between infection and dementia can be found in a report from pathologist Dr Alan MacDonald who examined brain biopsies from the McLean hospital (an affiliate of Harvard University) data bank from patients with confirmed Alzheimer’s disease.

His PCR analysis show that 7/10 of these patients had the DNA of Borrelia Bergdorferi in their brain, the ETO logic agent of lyme disease. We also fine at The majority of our chronically ill patients with lyme disease and co- infections have been exposed to high levels of heavy meals, such as Mercury and lead, and occasionally to aluminium. These also can cause memory and concentration problems, and can cause the production of elevated levels of free radicals, which can increase inflammation.

Similarly we are exposed to hundreds of environmental chemicals every day that are fat-soluble and therefore are deposited in brain. These can and do affect cognitive processing. We have enough causes for an epidemic of dementia in the general population. Drugs prescribed for Alzheimer’s only slow down the cognitive decline. Horowitz has seen the improvements in cognitive functioning after treating these patients for chronic tick borne infections, by  detoxifying them of fat-soluble toxins with glutathione, by using oral chelation agents to remove mercury lead and aluminium, and by identifying and treating B12 deficiencies and/or hypothyroidism.Horowitz screened 50 lyme patients for co- are infections with Ehrlichia, Babesia microti, Mycoplasma, and Bartonella henselae. He reported that treatment with two drugs was better than treatment with one drug, especially where intracellular bugs are concerned.

Horowitz tests for a broad range of co-infections including different strains of babesiosis, Ehrlichia, Anaplasma, Bartonella, Rickettsial infections such as Rocky Mountain spotted fever, Q fever, and typhus, tularemia, Brucella, chlamydia pneumonia, Mycoplasma species, viruses such as EBV, CMV, HHV 6, and parasites such as toxoplasmosis.

And increase in neuropsychiatric symptoms also takes place when a patient has contract in co- infections, such as a babesiosis, where Babesia  you can exacerbate underlying lyme disease symptoms including depression.

Other co- infections also can influence psychiatric symptoms. Ehrlichiosis  can cause central nervous system symptoms, as can viruses and intracellular infections with Mycoplasma spp. And Chlamydia pneumonia,  which are frequently found in MSIDS patients.

Often the patients with the worst neurological symptoms have lyme disease,  mycoplasma and/ Bartonella simultaneously, with or without the other co- infections . Bartonella henselae,  organism that causes cat Scratch fever, exacerbates many of the neurological and neuropsychiatric symptoms we see with lyme disease, and has been linked to anxiety disorders and depression, as well as various central nervous system abnormalities.

These include encephalomyelitis ( involving inflammation in both the brain and spinal cord, leading to difficulties with cognition and motor function) ;  transverse myelitis(inflammation and demyelination  of the spinal cord, leaving to difficulty walking);  spastic para paresis(stiffness and spasm in the lower extremities, affecting walking); seizures with hemiparesis (Weakness on one side of the body); cerebellar syndromes (Primarily defined by symptoms of dizziness and poor balance) and movement disorders (which can cause a variety of symptoms, including spasms twitching and involuntary movements).

Bartonella  can also be transmitted to the foetus.  Therefore resistant neuropsychiatric symptoms in children might be linked to maternal transmission of the organism, and should be suspected of the patient is living in a lyme endemic area or has cats at home.

Some patients with Bartonella have other severe neurologic manifestations with a ophthalmologic involvement i.e.  inflammation VI manifesting as optic neuritis, episcleritis, conjunctivitis, uveitis or iritis.

Bartonella  can also cause an oculoglandular  syndrome with preauricular adenopathy and conjunctivitis, neuroretinitis, branch retinal artery occlusion and vision loss. Bartonella should therefore be considered when patients present with particularly severe opthalmological  symptoms.

If we look at the symptom complex used to define CFS, we see that the symptoms overlap those seeing in lyme disease and associated co-infections and it explains why lyme disease is frequently misdiagnosed. Apart from the results of a spinal tap, one of the primary differences is the symptoms of persistent lyme disease tend to come and go, fluctuating with good and bad days, and the symptoms tend to  migrate.

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